Coronary Thrombosis is a narrowing or blockage of the arteries and vessels that provide oxygen and nutrients to the heart. It is caused by atherosclerosis, an accumulation of fatty materials on the inner linings of arteries. The resulting blockage restricts blood flow to the heart. When the blood flow is completely cut off, the result is a Heart Attack.

Coronary Thrombosis disease occurs when the coronary arteries become partially blocked or clogged. This blockage limits the flow of blood from the coronary arteries, which are the major arteries supplying oxygen-rich blood to the heart. The coronary arteries expand when the heart is working harder and needs more oxygen. Arteries would expand, for example, when a person is climbing stairs, exercising, or having sex. If the arteries are unable to expand, the heart is deprived of oxygen (myocardial ischemia). When the blockage is limited, chest pain or pressure, called angina, may occur. When the blockage cuts off the flow of blood, the result is heart attack (myocardial infarction or heart muscle death). Healthy coronary arteries are clean, smooth, and slick. The artery walls are flexible and can expand to let more blood through when the heart needs to work harder. The disease process in arteries is thought to begin with an injury to the linings and walls of the arteries. This injury makes them susceptible to atherosclerosis and blood clots (thrombosis). A heart attack (also called a myocardial infarction) is the death of part of the heart muscle due to its sudden loss of blood supply. Typically, the loss of blood supply is caused by a complete blockage of a coronary artery by a blood clot. A coronary artery is an artery that supplies blood (along with essential nutrients) to the heart muscle. Death of the heart muscle often causes chest pain and electrical instability of the heart muscle tissue. Electrical instability of the heart causes ventricular fibrillation (chaotic electrical disturbance). Orderly transmission of electrical signals in the heart is important for the regular beating (pumping) of the heart. A heart undergoing ventricular fibrillation simply quivers, and cannot pump or deliver oxygenated blood to the brain. Permanent brain damage and death can occur unless oxygenated blood flow is restored within five minutes. Approximately one million Americans suffer a heart attack annually. Four hundred thousand of these victims die as a result. Many of the heart attack deaths are due to ventricular fibrillation of the heart that occurs before the victim can reach any medical assistance or the emergency room. These electrical disturbances of the heart can often be successfully treated with medications or other means by paramedics in the “field,” or upon arrival to the hospital. Approximately 90% to 95% of heart attack victims who reach the hospital survive. The 5% to 10% who later die are those who have suffered major heart muscle damage, or who suffer an “extension” or enlargement of their heart attack. Early heart attack deaths can be avoided if a bystander starts CPR (cardiopulmonary resuscitation) within five minutes of the onset of ventricular fibrillation. CPR involves breathing for the victim and applying external chest compression to make the heart pump. When paramedics arrive, medications and/or electrical shock (cardioversion) to the heart can be administered to convert ventricular fibrillation to a normal heart rhythm. Therefore, prompt CPR and rapid paramedic response can improve the survival chances from a heart attack. Angina (Chest Pain) Angina (chest pain) is not a heart attack, but may be a warning of an impending heart attack. Angina also occurs when the heart muscle is not receiving enough oxygen. With angina, however, no permanent damage is done to the heart muscle. Angina is most commonly experienced with an activity such as shoveling snow, walking upstairs or uphill, consuming a large meal, or a stressful event. The symptoms of angina are similar to those of a heart attack. They are most commonly described as a squeezing, burning, tightness, fullness, or pressure across the chest. This discomfort may radiate to the shoulder, arms (especially the left), neck, jaw, teeth, earlobes, as well as the upper back between the shoulder blades. Numbness or tingling in the arms or hands may occur. Angina can be mistaken for indigestion. Unlike a heart attack, angina may be relieved by rest and/or nitroglycerin within 15 minutes. Both angina and heart attacks are caused by blockages in the coronary arteries. There are two main causes of clogged coronary arteries: Coronary Artery Disease   Coronary Thrombosis. See : Angina pectoris (Chest Pain) Treatment

Causes of Heart Attack

A heart attack is caused by the formation of a blood clot on a cholesterol plaque located on the inner wall of an artery to the heart (coronary artery). Cholesterol is a fatty chemical which is part of the outer lining of cells in the body. Cholesterol plaque is the formation of a hard, thick substance within the artery walls which is caused by deposits of cholesterol in the artery walls; a process that begins in the late teens. Over time, the accumulation of cholesterol plaque causes thickening of the artery walls and narrowing of the arteries; a process called atherosclerosis. Plaque accumulation can be accelerated by smoking, high blood pressure, elevated cholesterol, and diabetes. Ultimately, atherosclerosis causes significant narrowing of the coronary arteries. During exercise or excitement, the narrowed coronary arteries cannot increase the blood supply to meet the increased oxygen demand of the heart muscle. When the heart muscle is thus deprived of blood oxygen, a condition called ischemia results; ischemia may be associated with chest pain (angina pectoris). Typically, angina occurs with exertion, and subsides with rest. When the narrowing in the artery becomes critical, angina at rest or “unstable” angina may result. Unstable angina can be the harbinger of a heart attack in the near future.

Occasionally the surface of the cholesterol plaque in the artery may rupture, which leads to the formation of blood clot on the surface of the plaque, which then completely occludes blood flow in the vessel and results in a heart attack. The cause of this “plaque rupture” is largely unknown, but contributing factors may include cigarette smoking, elevated LDL cholesterol, elevated levels of blood catecholamines (adrenaline), high blood pressure, and other mechanical and biochemical forces.

Unlike exertional angina, death of the heart muscle from a heart attack is permanent. For more information, please visit the Cholesterol forum.

Chest pain or pressure is a common symptom of heart attack. Cardiac chest pain is often vague, or dull, and may be described as a pressure or band-like sensation, squeezing, heaviness, or other discomfort. Heart attacks frequently occur from 4:00 A.M. to 10:00 A.M. due to higher adrenaline amounts released from the adrenal glands during the morning hours. Increased adrenaline in the bloodstream can contribute to the above mentioned plaque rupture. Interestingly, heart attacks do not usually happen during exercise, although exercise is commonly associated with exertional angina. Approximately one quarter of all heart attacks are silent, without chest pain. In diabetics, the incidence of “silent” heart attacks may be much higher.

Heart attack victims may complain of:

·chest pressure
·sweating
·jaw pain
·heartburn and/or indigestion
·arm pain (more commonly the left arm, but may be either)
·upper back pain
·general malaise (vague feeling of illness)
·nausea
·shortness of breath

When severe chest pains, or any other of the symptoms mentioned above happen suddenly, then it could be a coronary thrombosis. If you are already suffering from angina pectoris and the prescribed western medicine does not seem adequate, then it might be a danger signal. Usually angina pectoris happens only after some sort of effort, and the physical effort required to bring it on is generally the same from day-to-day. An important warning sign of worsening atherosclerotic coronary disease is that the amount of effort required to bring on the angina begins to lessen. It is of great concern when the angina comes on with no effort at all. If angina suddenly happens at rest, and lasts longer than 10 minutes you must then come to Thetoleherbs KL Kuala Lumpur immediately or go to the nearest hospital. Thetoleherbs's WAY of Treatment Thetoleherbs' way can reverse the conditions with Thetoleherbs's Heart Herbal Medicine and it will dissolves any clots and arteries damage. It will strengthens the hearts Qi and its yin conditions. The Treatment takes between 2 weeks to 3 months for intensive Treatment to dissolve the blockage if there is no others complications. All these WITHOUT OPERATIONS its save and natural way of Thetoleherbs KL Kuala Lumpur. NO need for coronary angioplasty at all just our special heart herbal medicine since 145 years of Family research. 

Our Chinese Master has 145 YEARS (until 2006)OF RESEARCH adds to his OUTSTANDING RESULTS AND MIRACLE TREATMENT, FROM HIS INHERITATION (THE FOURTH GENERATION) and has conducted more than 100 seminars and trained more than 100 students from all over the world, we practice and produced acupuncture, herbal medicine, qi qong, food therapy and physiologic Treatment.

Those who cannot come to our centre for Treatment can e-mail to us giving all medical history and reports . Then we can send you the correct herbal medicine by courier service to your country after you have given us all your details. Then we will continue to guide you from here on your diet and other things. Many patients have been treatmentd with our e-herbal medicine without having to go for by-pass or surgery. 

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Feel FREE to enquire about yours or your friends health problems through e-mail. we can help to guild you from here.	Email
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